Thermo Fisher Scientific STK38L Polyclonal Antibody

Applications

Tested Dilution

Publications

Western Blot (WB)

1:1,000-1:3,000

-

Immunohistochemistry (Paraffin) (IHC (P))

1:50-1:200

-

Product Specifications

Species Reactivity

Human, Mouse, Rat

Host/Isotype

Rabbit / IgG

Class

Polyclonal

Type

Antibody

Immunogen

A synthesized peptide derived from human STK38L(Accession Q9Y2H1), corresponding to amino acid residues K403-G453. if (typeof window.$mangular === undefined || !window.$mangular) { window.$mangular = {}; } $mangular.antigenJson = \[{targetFamily:STK38L,uniProtId:Q9Y2H1-1,ncbiNodeId:9606,antigenRange:403-453,antigenLength:464,antigenImageFileName:PA5-104125_STK38L_Q9Y2H1-1_Rabbit.svg,antigenImageFileNamePDP:PA5-104125_STK38L_Q9Y2H1-1_Rabbit_PDP.jpeg,sortOrder:1}\]; $mangular.isB2BCMGT = false; $mangular.isEpitopesModalImageMultiSizeEnabled = true;

View immunogen .st0{fill:#FFFFFF;} .st1{fill:#1E8AE7;}

Conjugate

Unconjugated Unconjugated Unconjugated

Form

Liquid

Concentration

1 mg/mL

Purification

Affinity chromatography

Storage buffer

PBS, pH 7.4, with 50% glycerol

Contains

0.02% sodium azide

Storage conditions

-20°C

Shipping conditions

Wet ice

RRID

AB_2853453

Product Specific Information

Antibody detects endogenous levels of total STK38L and does not recognize STK38. The exact immunogen sequence is proprietary.

Target Information

STK38L (also known as NDR2) or Nuclear Dbf2-related protein is a serine/threonine kinase in the subgroup of the AGC kinase family. STK38L act as novel pro-apoptotic kinase that is activated by RASSF1A/MST1 in response to Fas receptor stimulation and promotes apoptosis. STK38L is mainly involved in the regulation of structural processes in differentiating and mature neuronal cells and it is highly expressed with highest levels observed in the thymus. STK38L contains 12 protein kinase catalytic subdomains and a potential bipartite nuclear localization signal.

For Research Use Only. Not for use in diagnostic procedures. Not for resale without express authorization.