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Thermo Fisher Scientific ORM1 Monoclonal Antibody (A4-G2-A3)
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Thermo Fisher Scientific ORM1 Monoclonal Antibody (A4-G2-A3)

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ORM1 단백질을 인식하는 Mouse IgG1 단클론 항체로, Western blot, IHC, Flow cytometry에 사용 가능. Human, Mouse, Rat 반응성. Protein G 정제 및 PBS/BSA/glycerol buffer 보존. 급성 염증 반응 연구 및 혈장 단백질 분석에 적합.

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MA532837
판매단위
pk
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마지막 업데이트 2025. 08. 04. 오전 04:48
Thermo Fisher Scientific MA532837 ORM1 Monoclonal Antibody (A4-G2-A3) 100 ul pk판매 단위 pk ·
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618,800원VAT 포함 680,680원

Thermo Fisher Scientific · Thermo Fisher Scientific ORM1 Monoclonal Antibody (A4-G2-A3)

Applications and Tested Dilutions

Application Tested Dilution
Western Blot (WB) 1:100–1:500
Immunohistochemistry (Paraffin) (IHC (P)) 1:50–1:100
Flow Cytometry (Flow) 1:50–1:1,000

Product Specifications

항목 내용
Species Reactivity Human, Mouse, Rat
Host / Isotype Mouse / IgG1
Class Monoclonal
Type Antibody
Clone A4-G2-A3
Immunogen Recombinant protein within Human Alpha-1-acid glycoprotein (aa 1–201)
Conjugate Unconjugated
Form Liquid
Concentration 2 mg/mL
Purification Protein G
Storage Buffer PBS, pH 7.4, with 0.2% BSA, 50% glycerol
Contains 0.05% sodium azide
Storage Conditions Store at 4°C short term. For long term storage, store at -20°C, avoiding freeze/thaw cycles.
Shipping Conditions Ambient (domestic); Wet ice (international)
RRID AB_2802484

Target Information

ORM1 (Alpha-1-acid glycoprotein) is an acute phase plasma protein synthesized by the liver. It regulates interactions between blood cells and endothelial cells and, together with haptoglobin and C-reactive protein, controls cell extravasation during infection and inflammation. ORM1 is a major acute phase reactant in both human and rat. In human plasma, ORM1 (44 kDa) is present at 0.5–1.4 mg/mL and increases during acute inflammation such as trauma, malignancies, myocardial infarction, rheumatoid arthritis, bacterial infections, surgery, or chronic pain. It is used as a marker for inflammatory conditions.


For Research Use Only. Not for use in diagnostic procedures. Not for resale without express authorization.

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