Thermo Fisher Scientific GABRA2 Monoclonal Antibody (N399/19), FITC
FITC 형광 표지된 GABRA2 단클론 항체로 인간, 마우스, 랫트 시료에 반응합니다. WB, IHC, ICC/IF에 사용 가능하며 단백질 G으로 정제되었습니다. 4°C 암소 보관하며, GABA-A 수용체 α2 서브유닛 검출에 적합합니다.
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Applications
- Western Blot (WB): 1:1,000
- Immunohistochemistry (IHC): Assay-dependent
- Immunocytochemistry (ICC/IF): 1:100
Product Specifications
| 항목 | 내용 |
|---|---|
| Species Reactivity | Human, Mouse, Rat |
| Host / Isotype | Mouse / IgG1 |
| Class | Monoclonal |
| Type | Antibody |
| Clone | N399/19 |
| Immunogen | Fusion protein amino acids 350–385 (Cytoplasmic C-terminus) of rat GABA-A Receptor Alpha2 |
| Conjugate | FITC |
| Excitation/Emission Max | 498 / 517 nm |
| Form | Liquid |
| Concentration | 1 mg/mL |
| Purification | Protein G |
| Storage Buffer | 9.1 mM sodium bicarbonate/PBS, pH 7.4, with 640.91 mM DMSO, 136.36 mM ethanolamine |
| Contains | No preservative |
| Storage Conditions | 4°C, store in dark |
| Shipping Conditions | Ambient (domestic); Wet ice (international) |
| RRID | AB_2932180 |
Additional Formats
- Unconjugated (MA5-27702)
- APC (MA5-45725)
- PE (MA5-45728)
- PerCP (MA5-45727)
- Request custom conjugation
Product Specific Information
A 1:100 dilution of MA5-45726 was sufficient for detection of GABA-A R, Alpha2 in 20 µg of mouse brain lysate by ECL immunoblot analysis using Goat anti-mouse IgG:HRP as the secondary antibody.
Detects approximately 55 kDa. Does not cross-react with GABA-A Receptor Alpha1.
This antibody was formerly sold as clone S399-19.
Target Information
GABA is the major inhibitory neurotransmitter in the mammalian brain where it acts at GABA-A receptors, which are ligand-gated chloride channels. Chloride conductance of these channels can be modulated by agents such as benzodiazepines that bind to the GABA-A receptor.
At least 16 distinct subunits of GABA-A receptors have been identified. Alternatively spliced transcript variants encoding the same protein have been found for this gene.
For Research Use Only. Not for use in diagnostic procedures. Not for resale without express authorization.
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