Thermo Fisher Scientific FLT1 Monoclonal Antibody (OTI11G5), TrueMAB
FLT1(VEGFR1)을 인식하는 mouse monoclonal antibody로, Western blot에 적합합니다. 인간 유래 단백질을 면역원으로 제작되었으며, 동결건조 형태로 제공됩니다. 친화 크로마토그래피로 정제되어 높은 순도를 보장합니다. 연구용으로만 사용 가능합니다.
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Applications and Tested Dilution
| Application | Tested Dilution |
|---|---|
| Western Blot (WB) | 1:2,000 |
Product Specifications
| 항목 | 내용 |
|---|---|
| Species Reactivity | Human |
| Host / Isotype | Mouse / IgG1 |
| Class | Monoclonal |
| Type | Antibody |
| Clone | OTI11G5 |
| Immunogen | Human recombinant protein fragment corresponding to amino acids 781–1338 of human FLT1 produced in HEK293T cells |
| Conjugate | Unconjugated |
| Form | Lyophilized |
| Concentration | 1 mg/mL |
| Purification | Affinity chromatography |
| Storage Buffer | PBS, pH 7.3, with 8% trehalose |
| Contains | No preservative |
| Storage Conditions | -20°C, Avoid Freeze/Thaw Cycles |
| Shipping Conditions | Ambient (domestic); Wet ice (international) |
Product Specific Information
For reconstitution, add 100 µL of distilled water to obtain a final antibody concentration of approximately 1 mg/mL.
For conjugation experiments using this carrier-free antibody, perform an additional desalting step (e.g., Zeba Spin Desalting Columns, 7K MWCO, 0.5 mL, Product #89882).
Target Information
FLT1 (VEGFR1) is a receptor tyrosine kinase involved in angiogenesis. It plays critical roles in both physiological and pathological processes such as tumor progression, diabetic retinopathy, rheumatoid arthritis, and wound healing.
FLT1 may act as a negative regulator of embryonic angiogenesis by inhibiting excessive endothelial cell proliferation. In adults, it can promote endothelial cell proliferation, survival, and angiogenesis in a cell-type-specific manner.
It has high affinity for VEGFA but relatively low kinase activity and may function as a negative regulator of VEGFA signaling by sequestering VEGFA and preventing its interaction with KDR.
For Research Use Only. Not for use in diagnostic procedures. Not for resale without express authorization.
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