Thermo Fisher Scientific HSP60 Monoclonal Antibody (4B9/89), Alexa Fluor 647
HSP60 단백질을 인식하는 Alexa Fluor 647 결합 단일클론 항체로, WB 및 ICC/IF에 적합합니다. 인간, 마우스, 비인간 영장류, 랫트에서 반응하며 고순도 Protein A 정제 제품입니다. 4°C에서 암소 보관하며 동결 금지.
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Thermo Fisher Scientific HSP60 Monoclonal Antibody (4B9/89), Alexa Fluor 647
Applications
Western Blot (WB)
Tested Dilution: Not specified
View 1 publicationImmunocytochemistry (ICC/IF)
Tested Dilution: 1–10 µg/mL
View references
Product Specifications
| 항목 | 내용 |
|---|---|
| Species Reactivity | Human, Mouse, Non-human primate, Rat |
| Published Species | Not Applicable |
| Host / Isotype | Mouse / IgG2a |
| Class | Monoclonal |
| Type | Antibody |
| Clone | 4B9/89 |
| Immunogen | Human placental HSP60 |
| Conjugate | Alexa Fluor™ 647 |
| Excitation / Emission Max | 650 / 671 nm |
| Form | Liquid |
| Concentration | 1 mg/mL |
| Purification | Protein A |
| Storage Buffer | PBS, pH 7.4, with 1 mg/mL BSA |
| Contains | 0.05% sodium azide |
| Storage Conditions | 4°C, store in dark, DO NOT FREEZE |
| Shipping Conditions | Ambient (domestic); Wet ice (international) |
| RRID | AB_2663058 |
Additional Formats
Product Specific Information
The MA3012A647 antigen is human placental HSP60.
Epitope mapping studies using human HSP60 deletion mutants suggest that this antibody binds either between amino acids 335–366 or 484–547.
Target Information
This gene encodes a member of the chaperonin family.
The encoded mitochondrial protein may function as a signaling molecule in the innate immune system and is essential for the folding and assembly of newly imported proteins in the mitochondria.
This gene is adjacent to a related family member, and the region between the two genes functions as a bidirectional promoter.
Several pseudogenes have been associated with this gene.
Two transcript variants encoding the same protein have been identified.
Mutations in this gene cause autosomal recessive spastic paraplegia 13.
For Research Use Only. Not for use in diagnostic procedures. Not for resale without express authorization.
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