Thermo Fisher Scientific MPZL1 Polyclonal Antibody
Thermo Fisher Scientific의 MPZL1 폴리클로날 항체는 인간, 마우스, 랫트에서 반응하며 Western blot 및 ELISA에 적합합니다. 재조합 단백질 항원을 기반으로 제작되었으며 고순도의 Affinity Chromatography로 정제되었습니다. PBS 및 글리세롤 버퍼로 안정화되어 -20°C에서 보관 가능합니다.
- 카탈로그번호
- PA588952
- 판매단위
- pk
카탈로그
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Applications
Western Blot (WB)
- Tested Dilution: 1:500–1:2,000
ELISA
- Tested Dilution: 1 µg/mL
Product Specifications
| 항목 | 내용 |
|---|---|
| Species Reactivity | Human, Mouse, Rat |
| Host / Isotype | Rabbit / IgG |
| Class | Polyclonal |
| Type | Antibody |
| Immunogen | Recombinant fusion protein containing amino acids 195–269 of human MPZL1 (NP_003944.1) |
| Conjugate | Unconjugated |
| Form | Liquid |
| Concentration | 1.95 mg/mL |
| Purification | Affinity Chromatography |
| Storage Buffer | PBS, pH 7.3, with 50% glycerol |
| Contains | 0.01% thimerosal |
| Storage Conditions | -20°C, Avoid Freeze/Thaw Cycles |
| Shipping Conditions | Wet ice |
| RRID | AB_2805248 |
Product Specific Information
- Immunogen sequence:
NSKRDYTGCS TSESLSPVKQ APRKSPSDTE GLVKSLPSGS HQGPVIYAQL DHSGGHHSDK INKSESVVYA DIRKN - Positive Samples: BxPC-3, HeLa, Mouse brain
- Cellular Location: Membrane, Single-pass type I membrane protein
Target Information
Cell surface receptor involved in signal transduction processes. Recruits PTPN11/SHP-2 to the cell membrane and is a putative substrate of PTPN11/SHP-2. Acts as a major receptor for concanavalin-A (ConA) and participates in ConA-induced cellular signaling, likely involving Src family tyrosine-protein kinases. Isoform 3 may have a dominant negative role by blocking tyrosine phosphorylation of MPZL1 induced by ConA. Isoform 1, but not isoform 2 or isoform 3, may regulate integrin-mediated cell motility.
⚠ WARNING: This product can expose you to chemicals including mercury, known to the State of California to cause birth defects or other reproductive harm.
For more information, visit www.P65Warnings.ca.gov.
For Research Use Only. Not for use in diagnostic procedures. Not for resale without express authorization.
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