Thermo Fisher Scientific IRF7 Polyclonal Antibody
IRF7 단백질을 표적하는 Thermo Fisher Scientific의 Rabbit Polyclonal Antibody. Western blot, IHC, ICC/IF, ELISA 등 다양한 응용 가능. Human, Mouse, Pig, Rat 반응성. 고순도 Protein A 정제, 안정적 액상 형태로 연구용에 적합.
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Applications and Tested Dilution
| Application | Tested Dilution |
|---|---|
| Western Blot (WB) | 1:500–1:2,000 |
| Immunohistochemistry (Paraffin) (IHC (P)) | 1:400 |
| Immunohistochemistry (Frozen) (IHC (F)) | 1:100–1:500 |
| Immunocytochemistry (ICC/IF) | 1:50–1:200 |
| ELISA | 1:500–1:1,000 |
Product Specifications
| Specification | Description |
|---|---|
| Species Reactivity | Human, Mouse, Pig, Rat |
| Host / Isotype | Rabbit / IgG |
| Class | Polyclonal |
| Type | Antibody |
| Immunogen | KLH conjugated synthetic peptide derived from human IRF7 (amino acids 401–503) |
| Conjugate | Unconjugated |
| Form | Liquid |
| Concentration | 1 mg/mL |
| Purification | Protein A |
| Storage Buffer | 0.01M TBS, pH 7.4, with 50% glycerol, 1% BSA |
| Contains | 0.02% ProClin 300 |
| Storage Conditions | -20°C |
| Shipping Conditions | Ambient (domestic); Wet ice (international) |
Target Information
Interferons (IFNs) are involved in numerous immune interactions during viral infections and play major roles in the induction and regulation of innate and adaptive antiviral mechanisms. During infection, host-virus interactions activate downstream molecules such as transcription factors IFN regulatory factor-3 (IRF3), which stimulate transcription of IFN-α/β genes. IRF7 contributes to transcriptional activation of virus-inducible cellular genes, including interferon beta chain genes.
IRF7 plays a major role in the innate immune pathway, interacting with Toll-like receptor (TLR) adaptor proteins MyD88 and Tirp/TRAM, functioning as an intermediate in TLR4 and TLR9 signaling.
There are at least four differentially spliced isoforms of IRF7, though their functions have not been clearly established.
For Research Use Only. Not for use in diagnostic procedures. Not for resale without express authorization.
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